Mechanisms by Which Garlic and Allyl Sulfur Compounds Suppress Cancer





J. A. Milner

Nutritional Science Research Group, Division Cancer Prevention, National Cancer Institute, National Institutes of Health, Department Health and Human Services, Rockville, MD 20892.



Garlic [Allium sativum] is among the oldest of all cultivated plants. Throughout history it has been revered for its medicinal properties with benefits proposed to encompass antimicrobial, antithrombotic, hypolipidemic, antiarthritic, hypoglycemic and antitumor effects. In some parts of the world the sales of garlic preparations ranks with those of leading prescription drugs. A variety of epidemiological and preclinical studies support the ability of allium foods, particularly garlic, to retard the cancer process. While epidemiological evidence suggests allium foods may retard the risk of cancer at several sites the strongest evidence exists for the gastrointestinal tract. It is unclear if the variability in epidemiological observations relates to the quantity, duration or to the type of garlic preparation provided or maybe to a modified response resulting from interactions with a host of dietary or environmental factors or with the consumers genetic (nutrigenetic) profile. Nevertheless, a recent report suggest that reduced risk of prostate cancer that was associated with enhanced allium vegetables consumption was independent of body size, intake of other foods, and total calorie intake. Fresh garlic extracts, deodorized garlic preparations, garlic oil and a number of individual organosulfur compounds derived from garlic have been found to modify one or more aspects of the cancer process in model systems. While the mechanism(s) by which garlic and its organosulfur constituents brings about these effects remains elusive several mechanisms are plausible. Among the possible mechanisms are alterations in drug metabolizing enzymes, reduced oxidative damage, suppressed tumor proliferation, enhanced apoptosis and suppressed angiogenesis. The shifts in carcinogen metabolism are frequently accompanied by a reduction in the activity of some cytochrome P450s enzymes, an increase in the activity of glutathione S-transferases carcinogens and an accompanying reduction in DNA adducts in several target tissues. Carcinogen induction studies reveal that the benefits of garlic are not limited to a specific species, to a particular tissue, or to a specific type of carcinogen. The antiproliferative activity associated with garlic and related organosulfur compounds has been observed in several tumor cell lines and may relate to changes in cell cycle regulation resulting from a reduction in p34(cdc2) kinase activity as a result of posttranslational changes in phosphorylation. The nutrigenomics effects of selected allyl sulfur compounds are evident by the widespread effects on the expressions of genes involved with various cancer processes. Biochemical analysis of tumors revealed several characteristic features of apoptosis, including the formation of DNA ladders, compaction of nuclear DNA, and the activation of caspase-3. Recently diallyl sulfide (DAS), an organosulfur compound present in garlic was reported to inhibit angiogenesis in tumor bearing mice. The exact molecular target for any of the alteration in the cancer process remains to be determined. Transgenic and knockout models may offer special insights into the sites of action of garlic and its allyl sulfur components. Overall, while compelling preclinical evidence supports garlic and its organic allyl sulfur components are effective inhibitors of the cancer process, considerably more controlled intervention studies in humans are needed to truly define who will benefit from garlic and its components and under what circumstances.




Key words: garlic, allyl sulfur, tumor, nutrigenomic, diet







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